Interview on Buteyko Breathing Method given to ANC news channel Philippines Nov 3, 2010.
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Air Date: May 2, 2011 -
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© 2011 SENS Foundation – www.sens.org The Fifth SENS conference – James Kirkland Author JL Kirkland Mayo Clinic Adipose tissue is at the nexus of processes involved in healthspan and metabolic dysfunction. Progression of age-related fat tissue dysfunction follows different trajectories across different fat depots, with fat becoming redistributed from subcutaneous to intra-peritoneal depots and ultimately ectopic sites, such as liver, muscle, and bone marrow. This is associated with insulin resistance, hypertension, atherosclerosis, strokes, myocardial infarction, cancer, and cognitive dysfunction. The preadipocytes from which new fat cells develop throughout the lifespan switch into a pro-inflammatory, tissue-remodeling state in old age, instead of differentiating into fat cells. In cellular senescence, proliferation becomes arrested and cells acquire a pro-inflammatory senescent secretory phenotype (SASP), with release of chemokines, cytokines, and extracellular matrix proteases. We found that chronological aging, obesity, progeroid states, and serial subculturing result in accumulation of senescent preadipocytes with impaired adipogenesis and a pro-inflammatory phenotype. Senescent cells accumulate to a greater extent in subcutaneous than intraperitoneal fat. TNF alpha and IL-6, which increase in fat tissue with aging and obesity, can induce preadipocyte cellular senescence and pro-inflammatory cytokine and chemokine secretion, potentially spreading senescence from cell … -
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